Nicotinic Acid, tablets (prolonged release), 500 mg, 750 mg and 1 g, Niaspan®, July 2006
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Public Summary Document
Product: Nicotinic Acid, tablets (prolonged release), 500 mg, 750 mg and 1 g, Niaspan®
                           Sponsor: Alphapharm Pty Ltd
                           Date of PBAC Consideration: July 2006
                            
                        
1. Purpose of Application
This submission sought the listing of a prolonged release formulation of nicotinic
                           acid as a restricted benefit for use in combination with an HMG CoA reductase inhibitor
                           (statin) in Type II diabetic patients with dyslipidaemia whose High Density Lipoprotein
                           Cholesterol (HDL-C) levels are inadequately controlled despite monotherapy with a
                           statin.
                            
                        
2. Background
This formulation of nicotinic acid had not previously been considered by the PBAC.
                           
                           The unrestricted listing for nicotinic acid immediate release tablets 250 mg was deleted
                           from the PBS at the request of the sponsor on 1 December 2005. However, a non-subsidised
                           250 mg tablet remains available.
                            
                        
3. Registration Status
The TGA registered Niaspan on the 27 February 2006 for “the treatment of mixed dyslipidaemia,
                           and primary hypercholesterolaemia, as adjunctive therapy to diet. Prior to initiating
                           therapy with nicotinic acid, secondary causes of hypercholesterolaemia (e.g. poorly
                           controlled diabetes mellitus, hypothyroidism, nephrotic syndrome, dysproteinaemias,
                           obstructive liver disease, other drug therapy, alcoholism) should be identified and
                           treated.”
                            
                        
4. Listing requested and PBAC’s View
The submission requested a restricted benefit for use in combination with a HMG CoA
                           reductase inhibitor (statin) in type 2 diabetic patients with dyslipidaemia whose
                           HDL-C levels are inadequately controlled despite monotherapy with a statin. Inadequate
                           control is defined as HDL-C<1 mmol/L after at least 3 month’s treatment with a statin.
                           
                           The PBAC’s view was that an authority required restriction would be more appropriate
                           to ensure that the stated objectives of the proposed indication were achieved. The
                           Pre-PBAC response clarified that the sponsor’s intention was to seek listing as an
                           add-on treatment in ‘at-risk’ patients.
                            
                        
5. Clinical place for the proposed therapy
Niaspan will provide add-on treatment for Type 2 diabetic patients with dyslipidaemia
                           whose HDL-C levels are inadequately controlled with a statin alone.
                            
                        
6. Comparator
The submission nominated ezetimibe as the main comparator.
                           
                           The selection of ezetimibe as the comparator in the submission was not accepted by
                           the PBAC. The PBAC considered nicotinic acid will not replace ezetimibe in clinical
                           practice. It represents a new pharmacological action, not currently available to prescribers
                           under the PBS. The Committee considered the comparator could be placebo, as an add-on
                           to a statin, if the objective of therapy with nicotinic acid is improvement in overall
                           cardiovascular risk, as well as standard-release nicotinic acid (not on PBS).
                            
                        
7. Clinical Trials
The scientific basis of comparison was an indirect comparison of nicotinic acid prolonged release tablets and ezetimibe based on results from:
- one randomised trial of prolonged-release nicotinic acid tablets versus placebo over 16 weeks in patients with type 2 diabetes with dyslipidaemia treated with/without a statin (Protocol MA-98-0108, published as Grundy et al, 2002); and
- one randomised trial comparing the addition of ezetimibe to statin therapy with a doubling of the statin dose over 24 weeks in patients in patients with type 2 diabetes and dyslipidaemia treated with a statin (Gaudiani et al 2005).
The trial published at the time of the submission were as follows:
                            
                        
| Trial/First author | Publication title | Citations | 
|---|---|---|
| Protocol MA-98-0108/Grundy SM | Efficacy, safety and tolerability of once-daily niacin for the treatment of dyslipidaemia associated with type 2 diabetes | Arch Intern Med. 2002; 162:1568-76. | 
| Gaudiani et al. | Efficacy and safety of ezetimibe co-administered with simvastatin in thiazolidinedione-treated type 2 diabetic patients | Diabetes Obes Metab 2005; 7: 88-97. | 
8. Results of Trials
According to the analyses comparing nicotinic acid + statin (ie, the subgroup of patients in Protocol MA-98-0108) and ezetimibe 10 mg + simvastatin 20 mg (ie, patients in Gaudiani et al, 2005 trial), there appeared to be:
- numerically larger increase in HDL-C from baseline and numerically larger decrease in triglyceride from baseline favouring nicotinic acid over ezetimibe;
- numerically larger decrease in LDL-C and total cholesterol from baseline favouring ezetimibe over nicotinic acid;
- numerically larger decrease in TC: HDL-C ratio from baseline favouring nicotinic acid over ezetimibe; and
- no effect on LDL-C due to nicotinic acid.
The PES Commentary advised, the drawing of conclusions based on the above data was
                           problematic due to the lack of a common reference across the trials and due to potential
                           differences in the baseline characteristics of patients recruited to each trial.
                           
                           A matter of concern was the worsening of glycaemic control associated with the high
                           dose nicotinic acid. According to the analyses, greater increases from baseline were
                           generally observed for both HbA1c and fasting blood glucose in the nicotinic acid
                           + statin subgroup in Protocol MA-98-0108 compared to the ezetimibe + simvastatin group
                           in Gaudiani et al (2005), though generally not statistically significant.
                           
                           In Protocol MA-980108, the proportion of patients experiencing flushing were reported
                           as 5/45 (11.1%) for placebo, 30/45 (66.7%) for nicotinic acid 1,000mg and 35/52 (67.3%)
                           for nicotinic acid 1,500mg. The maximum intensity of flushing events was generally
                           mild to moderate in severity. No cases of myopathy were reported in Protocol MA-98-0108
                           and Gaudiani et al (2005). There were no other important differences in safety parameters
                           across the two trials.
                            
                        
9. Clinical Claim
The implicit claim in the submission was that nicotinic acid had advantages in terms
                           of effectiveness over ezetimibe and was associated with a similar safety profile.
                           
                           For PBAC’s view see Recommendation and Reasons.
10. Economic Analysis
A preliminary economic evaluation was presented. The choice of the cost-effectiveness
                           approach was valid only if the PBAC accepted the submission’s implicit claim that
                           nicotinic acid had advantages in terms of effectiveness (in terms of effect on TC:
                           HDL-C ratio) over ezetimibe. The resources included were drug costs.
                           
                           The trial-based incremental cost per extra point reduction in TC: HDL-C ratio was
                           dominated by nicotinic acid which was claimed to be less expensive and more effective
                           than ezetimibe.
                           
                           A modelled economic evaluation against placebo was presented. The base case modelled
                           incremental discounted cost per extra QALY gained was either < $15,000 or in the range
                           $15,000 - $45,000 over 52, or 44 years, for nicotinic acid 1,000 mg/day vs placebo,
                           depending on the model used (model based on risk equations from UK Prospective Diabetes
                           Study (UKPDS) or from Framingham Heart Study (FHS), respectively); < $ 15,000 over
                           52, or 44 years, for nicotinic acid 1,500 mg/day vs placebo, depending on the model
                           used (model based on risk equations from UKPDS or from FHS, respectively)
                            
                        
11. Estimated PBS Usage and Financial Implications
The likely number of patients per year was > 200,000 in 2010, while the financial
                           cost per year to the PBS (excluding co-payments) was < $10 million in 2010.
                            
                        
12. Recommendation and Reasons
The PBAC considered an authority required restriction would be more appropriate to
                           ensure that the stated objectives of the proposed indication are achieved.
                           
                           The selection of ezetimibe as the comparator in the submission was not accepted by
                           the PBAC. The PBAC considered nicotinic acid will not replace ezetimibe in clinical
                           practice. It represents a new pharmacological action, not currently available to prescribers
                           under the PBS. The Committee considered the comparator could be placebo, as an add-on
                           to a statin, if the objective of therapy with nicotinic acid is improvement in overall
                           cardiovascular risk, as well as standard-release nicotinic acid (not on PBS).
                           
                           The Pre-PBAC response clarified that the intent was to seek listing as an add-on treatment
                           in ‘at-risk’patients.
                           
                           More importantly, the PBAC did not consider the submission’s implicit claim that nicotinic
                           acid prolonged release tablets are more effective (in terms of effect on TC: HDL-C
                           ratio) and no worse in terms of safety than ezetimibe, was reasonable. Nicotinic acid
                           when used as in the trials presented , had no effect on LDL-C and only a small effect
                           on triglycerides but only at the higher dose. In addition, there is no clinical trial
                           evidence demonstrating that raising HDL-C improves clinical outcomes. Therefore, the
                           validity of raising HDL-C as a surrogate outcome has not been established. All other
                           lipid-lowering drugs already listed on the PBS had either clinical outcomes data and/or
                           evidence of lowering LDL-C, hence TC:HDL ratio. The primary mechanism by which nicotinic
                           acid lowers the TC:HDL ratio is via HDL-C rather than LDL-C. The impact of this different
                           mechanism on cardiovascular events is unknown. This is also a fundamental concern
                           with Framingham based modelled economic evaluation which has the TC:HDL ratio as a
                           driver.
                           
                           The PBAC noted the Pre-PBAC Response advice that overall it has been concluded from
                           prospective population studies that for every 0.025 mmol/L increase in HDL-C, cardiovascular
                           risk is reduced by 2 to 5%. However, the Committee considered that this clinical benefit
                           seen in epidemiological data could not be assumed to flow from raising HDL-C with
                           a drug.
                           
                           The PBAC noted the trial evidence presented showed no effect was seen on LDL with
                           nicotinic acid in this setting (ie with background statin therapy), whereas it had
                           a significant TC-lowering effect (10%), in the Coronary drug project: experience with
                           niacin (Coronary Drug Project Research Group. Berge KG, Canner PL. Eur J Clin Pharm
                           40 Supp 1: S49-51; 1991).
                           
                           There was concern that the high doses of nicotinic acid worsen both HbA1c and fasting
                           blood sugar levels. The Pre-PBAC response, while noting that similar outcomes were
                           observed in the Gaudiani trial for the ezetimibe and simvastatin arm, and the simvastatin
                           40 mg arm respectively, pointed out that in terms of HBA1c there was a statistically
                           significant difference only in the 1500 mg nicotinic acid arm (p=0.047) in the ITT
                           analysis (MA 98-0108).
                           
                           Given the PBAC’s views in relation to the comparator and the implicit clinical claim,
                           the economic evaluation was not pivotal to its recommendation. The preliminary economic
                           evaluation was not relevant. However, the PBAC considered the placebo based models
                           would have been acceptable if based on appropriate clinical data. Specific comments
                           with respect to the two models presented included:
                        
- The second model was based on the UKPDS risk equation which overestimates the baseline risk in the Australian population.
- The acceptability of the assumption in the model of differences in baseline parameters for patients in the two arms of the model based on non-significant differences in the trial. It was unclear whether the model-derived differences in effectiveness and cost-effectiveness are driven by differences in baseline characteristics or by true treatment differences.
- The use in the UKPDS model of EQ5D utility decrements may overestimate the true quality of life decrements because of the assumption of an additive effect of multiple complications and because a feature of the EQ5D instrument is that because it has relatively few dimensions and levels to describe health states, when a difference is detected, the numerical value can appear disproportionately large.
The PBAC therefore rejected the submission on the basis the comparator was not appropriate
                           and lack of clinical evidence to support the implicit claim that an increase in HDL
                           levels reduces cardiovascular risk.
                            
                        
13. Context for Decision
The PBAC helps decide whether and, if so, how medicines should be subsidised in Australia.
                           It considers submissions in this context. A PBAC decision not to recommend listing
                           or not to recommend changing a listing does not represent a final PBAC view about
                           the merits of the medicine. A company can resubmit to the PBAC or seek independent
                           review of the PBAC decision.
                            
                        
14. Sponsor’s Comment
Alphapharm looks forward to working with the PBAC to address the issues raised by the Committee.




